By Barry Popkin (auth.), Michael Freemark (eds.)
Childhood weight problems and its co-morbidities -- together with style 2 diabetes, high blood pressure, dyslipidemia, sleep apnea, and fatty liver ailment -- have noticeable impressive raises in recent times. regardless of a wealth of research, there's substantial controversy in regards to the etiology of adolescence weight problems and the optimum ways for prevention and remedy. Pediatric weight problems: Etiology, Pathogenesis, and remedy addresses the talk with more than a few good points that make it a different source when you take care of overweight youngsters and their households. Written from a viewpoint that's foreign in scope, the prestigious authors re-assess the jobs of genetic and environmental components within the pathogenesis of youth weight problems and severely evaluation new reviews of the results of way of life, pharmacologic, and surgical interventions. The evidence-based technique of Pediatric weight problems: Etiology, Pathogenesis, and remedy offers a finished and beneficial consultant for all healthcare services fascinated about the evaluate and care of youngsters with dietary and metabolic illness and with the societal implications of the weight problems epidemic.
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Additional info for Pediatric Obesity: Etiology, Pathogenesis, and Treatment
1999;48:347–52. 30. Barr VA, Malide D, Zarnowski MJ, Taylor SI, Cushman SW. Insulin stimulates both leptin secretion and production by white adipose tissue. Endocrinology. 1997;138:4463–72. 31. Kolaczynski JW, Nyce MR, Considine RV, Boden G, Nolan JJ, Henry R, Mudaliar SR, Olefsky J, Caro JF. Acute and chronic effects of insulin on leptin production in humans: studies in vivo and in vitro. Diabetes. 1996;45:699–701. 32. Singhal A, Farooqi IS, O’Rahilly S, Cole TJ, Fewtrell M, Lucas A. Early nutrition and leptin concentrations later in life.
J Pediatr. 2006;148:23–9. 147. Mietus-Snyder ML, Lustig RH. Childhood obesity: adrift in the “limbic triangle”. Annu Rev Med. 2008;59:119–34. III THE GENETICS OF CHILDHOOD OBESITY 3 Monogenic Obesity David Meyre and Philippe Froguel CONTENTS I NTRODUCTION M ONOGENIC O BESITY C AUSED BY M UTATIONS IN THE L EPTIN /M ELANOCORTIN PATHWAY II-M ONOGENIC O BESITY WITH N EUROLOGICAL F EATURES III-A C ONTINUUM B ETWEEN M ONOGENIC AND P OLYGENIC O BESITY ? 12 billion people by 2030 (1). The epidemic of obesity is attributed to recent changes in the environment (easy access to highenergy palatable food, combined with a decreased physical activity), whereas individual differences in obesity risk are attributed to genetic differences between individuals: the heritability of body mass index (BMI) has been estimated to approximate 70% in adults and is even higher (77%) for younger people raised in an increasingly obesogenic environment (2).
Nat Genet. 1997;17:273–274. 67. Wellman PJ. Modulation of eating by central catecholamine systems. Curr Drug Targets. 2005;6:191–99. 68. Leibowitz S, Roosin P, Rosenn M. Chronic norepinephrine injection into the hypothalamic paraventricular nucleus produces hyperphagia and increased body weight in the rat. Pharmacol Biochem Behav. 1984;21:801–8. 69. Wellman PJ, Davies BT. Reversal of cirazoline-induced and phenylpropanolamine-induced anorexia by the alpha-1receptor antagonist prazosin. Pharmacol Biochem Behav.