Painful Diabetic Neuropathy in Clinical Practice by Andrew J. M. Boulton, Loretta Vileikyte

By Andrew J. M. Boulton, Loretta Vileikyte

Painful Diabetic Neuropathy in medical perform presents a concise, didactic and crucial source for clinicians within the administration of neuropathic soreness in diabetic patients.

This quantity opens with overviews of epidemiology, category and scientific good points, together with a dialogue of the detrimental impact of painful neuropathic signs on caliber of lifestyles. those are via a bankruptcy on prognosis and staging, consisting of ways to historical past taking, scientific exam, soreness evaluate scales, trying out and staging. The ebook concludes with a bankruptcy at the quite a few techniques within the administration of neuropathic ache, together with the main up to date guidance at the pharmacological therapy of this condition.

This concise instruction manual is a useful reference for fundamental care practitioners and diabetologists who desire to sustain to this point with the analysis and administration of neuropathic soreness.

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Thus, these data and data from genetically altered mice suggest that while impaired glucose uptake is a critical factor in the development of type 2 diabetes, the insulin-stimulated component of glucose uptake in muscle may not be as important as the contraction/exercise-stimulated component of glucose uptake. This analysis is consistent with the dramatic improvement in glucose tolerance observed with exercise in animals and humans with type 2 diabetes. These observations fit well with the notion that the liver (not muscle) controls minute-to-minute glucose levels and that muscle contraction is responsible for a large component of glucose disposal into muscle.

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1995). Thus, these data and data from genetically altered mice suggest that while impaired glucose uptake is a critical factor in the development of type 2 diabetes, the insulin-stimulated component of glucose uptake in muscle may not be as important as the contraction/exercise-stimulated component of glucose uptake. This analysis is consistent with the dramatic improvement in glucose tolerance observed with exercise in animals and humans with type 2 diabetes. These observations fit well with the notion that the liver (not muscle) controls minute-to-minute glucose levels and that muscle contraction is responsible for a large component of glucose disposal into muscle.

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