Frontiers in Gynecological Endocrinology: Volume 3: Ovarian by Andrea R. Genazzani, Basil C. Tarlatzis

By Andrea R. Genazzani, Basil C. Tarlatzis

This quantity represents an up to date review on ovarian capabilities and copy, delivering the reader with the most recent advances in gynecological endocrinology. It is released in the overseas Society of Gynecological Endocrinology (ISGE) sequence, and relies at the 2015 foreign university of Gynecological and Reproductive Endocrinology iciness path. The booklet covers a truly wide selection of topics with specific concentration on ovulation and assisted replica, ovarian getting older and fertility, and untimely ovarian failure. The volume will be an invaluable tool for gynecologists, endocrinologists, obstetrician, and specialists in women’s well-being.

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19] showing that growth hormone as adjuvant treatment of ovarian stimulation increased the pregnancy rate in patients with low response, many authors published their experience with varying results. 6. We found no differences in response, or in efficacy in pregnancy term, in both groups. 5). 3 Oocyte Accumulation We would like to mention our experience in oocyte accumulation in patients undergoing ovarian stimulation for preimplantation genetic screening (PGS) who presented insufficient response.

C. G. Artini the role of the oocyte extends far beyond its functions in the transmission of genetic information and supply of raw materials to the early embryo. It also has a critical part to play in mammalian follicular control and the regulation of oogenesis, ovulation rate, and fecundity [3, 4]. This chapter is aimed to explore the GCs gene activity in physiological and pathological conditions. 2 Gene Modulation of Granulosa Cells During Folliculogenesis The ovarian follicle development is a complex process involving the coordination of many factors that regulate the growth and differentiation of the female gamete and the surrounding somatic components.

According to Tapia et al. [21], at the secretory phase in the human endometrium, the mRNA levels of the complement system molecules C4b-binding protein (C4BP) and adipsin (complement component factor D, CFD) increase. It is postulated that the complement system might provide the uterine cavity with immunity against bacterial infection. In this sense, C4BP may protect the embryo where an increased expression of an inhibitor of complement system activation could reduce the chance of a misdirected complement attack to the embryo [21].

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