38th International Symposium on Growth Hormone and Growth by H. P. F. Koppeschaar, Torsten Tuvemo, Peter Trainer, Philip

By H. P. F. Koppeschaar, Torsten Tuvemo, Peter Trainer, Philip Zeitler

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Alendronate), would it be better to provide intermittent treatment? Finally, should we monitor therapy? In clinical trials, it is likely that subjects have a favourable reduction in fracture risk. However, in clinical practice it is possible that not all subjects benefit from treatments either because they do not fully comply with dosing instructions or because they have some coexistent disease. Treatment can be monitored by measuring bone mineral density. Bone mineral density of the spine is usually measured after 1–2 years, with an increase of more than 3–6% probably indicating a favourable response.

This ultimately translates into protein sparing by impeding the demand for glucose oxidation and thus gluconeogenesis from protein. any significant suppressive effect of GH on PI3 kinase activity in normal subjects despite the fact that GH significantly stimulated lipolysis and induced insulin resistance [11]. , unpublished data]. matter. Growth hormone is usually administered as daily subcutaneous injections in the evening, and the dose is tailored to maintain insulin-like growth factor I levels within the normal range for age and sex [12].

Based on the study by Nielsen and associates, elevated FFA levels play a causal role [10]; however, there is no experimental human in vivo data to support the belief that insulin-signaling pathways are affected [11]. Many more studies are needed with particular attention focused on the dosing and timing of GH and insulin and the optimal time to obtain a muscle biopsy. It is also likely that newer methods such as gene arrays and proteomics may be important supplements to current assays, which only measure the activity of single proteins.

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